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Traumatic cardiac and mechanical disorders are structural or functional injuries to the heart and great vessels resulting from blunt or penetrating trauma, or from iatrogenic/device-related causes. These conditions include myocardial contusion, chamber rupture, pericardial tamponade, valvular disruption, septal defects, coronary injury, and prosthesis/device complications. They represent life-threatening emergencies requiring rapid recognition and intervention.
By Mechanism
Blunt Cardiac Injury (BCI): Myocardial contusion, chamber rupture, coronary artery injury, valvular disruption.
Penetrating Cardiac Trauma: Stab wounds, gunshot wounds, iatrogenic perforation.
Mechanical Complications: Pericardial tamponade, acute severe regurgitation, traumatic VSD, papillary muscle rupture.
Great Vessel Trauma: Traumatic aortic rupture, pulmonary artery/vein laceration, SVC/IVC injury.
Device/Prosthesis-Associated: Pacemaker lead perforation, LVAD trauma, prosthetic valve disruption.
Blunt trauma: Rapid deceleration or direct chest compression → myocardial bruising, coronary dissection, chamber rupture.
Penetrating trauma: Direct laceration or perforation of myocardium/pericardium → hemorrhage, tamponade.
Mechanical complications: Secondary disruption of valves, septum, or pericardium → acute hemodynamic instability.
Great vessel trauma: Aortic isthmus rupture or venous tears → rapid exsanguination.
Device trauma: Malposition or migration causing perforation or obstruction.
Accounts for up to 20% of fatalities in blunt chest trauma.
Penetrating cardiac trauma: Mortality >50% pre-hospital; survival depends on rapid surgical access.
Most common in young adults due to high-energy mechanisms (MVCs, gunshot wounds).
Blunt trauma: MVC, falls, sports injuries, crush injuries.
Penetrating trauma: Firearm wounds, stab injuries, surgical/iatrogenic injury.
Iatrogenic: Catheter perforation, pacemaker/ICD leads, ablation, central line placement.
Device-related: LVAD malposition, prosthetic valve disruption.
High-speed collisions
Chest wall deformities (sternal fractures, flail chest)
Anticoagulation (exacerbates bleeding)
Prior cardiac surgery (adhesions predispose to injury)
Implanted devices
Chest pain (sharp, crushing, pleuritic, or stabbing)
Dyspnea or orthopnea
Palpitations, syncope
Fatigue, dizziness (low output state)
History of recent trauma (MVC, fall, penetrating injury)
Vital Signs
Hypotension (shock from tamponade, rupture, or hemorrhage)
Tachycardia (compensatory or arrhythmia-related)
Bradycardia (conduction block or vagal response)
Pulsus paradoxus (tamponade)
General Appearance
Acute distress, diaphoresis
Cyanosis or pallor
Respiratory distress (tachypnea, accessory muscle use)
External trauma: ecchymosis, abrasions, “seatbelt sign,” rib or sternal tenderness
Cardiovascular
Jugular venous distension (tamponade, right heart failure)
Muffled or distant heart sounds (tamponade)
New murmurs (valvular rupture, traumatic VSD)
Pericardial rub (traumatic pericarditis)
Irregular rhythm on auscultation (PVCs, AF, complete heart block)
Precordial tenderness/crepitus
Respiratory
Decreased breath sounds (hemothorax, pneumothorax)
Tracheal deviation (tension pneumothorax or massive hemopericardium)
Rales (pulmonary edema from acute MR or LV failure)
Abdominal
Distension from hepatic congestion (tamponade, right heart failure)
Associated visceral trauma
Extremities
Cool, clammy skin (shock)
Weak or absent peripheral pulses (aortic injury, hypoperfusion)
Edema (delayed right-sided injury sequelae)
Acute coronary syndrome
Aortic dissection
Pulmonary embolism
Pneumothorax/hemothorax
Myocardial infarction without trauma
Cardiac tamponade of non-traumatic etiology
Myocarditis
ECG: Arrhythmias, ST-T changes, conduction delays
Chest X-ray: Widened mediastinum (aortic rupture), cardiomegaly (tamponade), rib/sternal fractures
Echocardiography (TTE/TEE): Pericardial effusion, tamponade, chamber rupture, valvular injury
Troponin: Elevated in myocardial contusion or coronary injury
CT Angiography: Aortic/great vessel evaluation; coronary artery trauma
Cardiac MRI: Myocardial contusion or coronary dissection in stable patients
Cardiac catheterization: Diagnostic/therapeutic in coronary injuries
Pericardiocentesis fluid analysis: If effusion present
Initial stabilization (ATLS principles): Airway, breathing, circulation
IV fluids and blood products for hemorrhagic shock
Oxygen if hypoxemic
Arrhythmia management: ACLS algorithms, temporary pacing for high-degree AV block
Anticoagulation: Generally avoided unless clear coronary thrombosis without bleeding risk
Pericardial tamponade: Emergent pericardiocentesis or thoracotomy
Chamber or valvular rupture: Immediate surgical repair
Coronary artery injury: PCI or CABG
Great vessel trauma: Open surgical repair or endovascular stenting
Device-related injury: Lead revision, LVAD repositioning, valve re-replacement
Education: Nature of traumatic cardiac injury, need for close monitoring, risk of late arrhythmias or heart failure
Lifestyle: Avoid strenuous activity during recovery; follow post-surgical precautions
Screening: Serial ECGs, echocardiograms for arrhythmia or structural changes
Vaccinations: Influenza and pneumococcal vaccination for those with chronic cardiac dysfunction
Cardiothoracic Surgery: For repair of ruptures, valve injury, great vessel trauma
Cardiology: Ongoing management of myocardial contusion, coronary trauma, arrhythmias
Critical Care/Trauma Surgery: Initial stabilization and ICU management
Rehabilitation/Physical Therapy: Recovery post-surgery or hospitalization
Short-term: Monitor for arrhythmias, effusion re-accumulation, heart failure
Intermediate: Echocardiography at 2–6 weeks; stress testing if coronary injury suspected
Long-term: Lifelong follow-up in survivors of major trauma for late complications (HF, aneurysm, arrhythmia)
HMD is a beacon of medical education, committed to forging a global network of physicians, medical students, and allied healthcare professionals.